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Epigenetic factor HDAC6 has been reported to associate tightly with cell autophagy among all histone deacetylases. Previous study revealed that HDAC6 mediated retrograde transport of inclusion bodies containing aggregate to be degraded by autophagy through deacetylating ɑ-tubulin. In line with our study, HDAC6 levels were increased in retinas of diabetic rats compared with healthy rats, which were associated with autophagy induced by oxidative stress injury, was attenuated by GLP-1treatment. To our best knowledge, it is the first report that the protective effects of GLP-1against DR may be through modulating HDAC6 expression..

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